Supervisor：Ministry of Education of the People's Republic of ChinaSponsor：Sichuan University Editor-in-chief：ZHOU DONG Publishing period：
Bimonthly
ISSN：2096-0247CN：51-1762/R
Lancaster E, Lai M, Peng X, et al. Antibodies to the GABA(B) receptor in limbic encephalitis with seizures: case series and characterisation of the antigen. Lancet Neurology, 2010, 9(1): 67-76.
3.
Poliak S, Gollan L, Martinez R, et al. Caspr2, a new member of the neurexin superfamily, is localized at the juxtaparanodes of myelinated axons and associates with K+ channels. Neuron, 1999, 24(4): 1037-1047.
4.
Lu Z, Reddy M, Liu J, et al. Molecular architecture of contactin-associated protein-like 2 (CNTNAP2) and its interaction with contactin 2 (CNTN2). Journal of Biological Chemistry, 2016, 291(46): 24133-24147.
5.
Sonderen AV, Schreurs M, Wirtz PW, et al. From VGKC to LGI1 and Caspr2 encephalitis: The evolution of a disease entity over time. Autoimmunity Reviews, 2016, 15(10): 970-974.
6.
P Jesús, Frank L, Francesco M, et al. Human N-methyl D-aspartate receptor antibodies alter memory and behaviour in mice. Brain, 2015, 138(1): 94-109.
7.
V Sonderen, Agnes, Arino, et al. The clinical spectrum of Caspr2 antibody-associated disease. Neurology, 2016, 87(18): 521-522.
8.
Sonderen AV, Petit-Pedrol M, Dalmau J, et al. The value of LGI1, Caspr2 and voltage-gated potassium channel antibodies in encephalitis. Nature Reviews Neurology, 2017, 13(5): 290-301.
9.
Gadoth A, Pittock SJ, D Dubey, et al. Expanded phenotypes and outcomes among 256 LGI1/CASPR2-IgG positive patients. Annals of Neurology, 2017, 82(1): 79-92.
10.
Patterson KR, Dalmau J, Lancaster E. Mechanisms of Caspr2 antibodies in autoimmune encephalitis and neuromyotonia. Annals of Neurology, 2018, 83(1): 40-51.
11.
Bien CG, Mirzadjanova Z, Baumgartner C, et al. Anti‐contactin‐associated protein‐2 encephalitis: relevance of antibody titres, presentation and outcome. European Journal of Neurology, 2016, 24(1): 175-186.
2. Lancaster E, Lai M, Peng X, et al. Antibodies to the GABA(B) receptor in limbic encephalitis with seizures: case series and characterisation of the antigen. Lancet Neurology, 2010, 9(1): 67-76.
3. Poliak S, Gollan L, Martinez R, et al. Caspr2, a new member of the neurexin superfamily, is localized at the juxtaparanodes of myelinated axons and associates with K+ channels. Neuron, 1999, 24(4): 1037-1047.
4. Lu Z, Reddy M, Liu J, et al. Molecular architecture of contactin-associated protein-like 2 (CNTNAP2) and its interaction with contactin 2 (CNTN2). Journal of Biological Chemistry, 2016, 291(46): 24133-24147.
5. Sonderen AV, Schreurs M, Wirtz PW, et al. From VGKC to LGI1 and Caspr2 encephalitis: The evolution of a disease entity over time. Autoimmunity Reviews, 2016, 15(10): 970-974.
6. P Jesús, Frank L, Francesco M, et al. Human N-methyl D-aspartate receptor antibodies alter memory and behaviour in mice. Brain, 2015, 138(1): 94-109.
7. V Sonderen, Agnes, Arino, et al. The clinical spectrum of Caspr2 antibody-associated disease. Neurology, 2016, 87(18): 521-522.
8. Sonderen AV, Petit-Pedrol M, Dalmau J, et al. The value of LGI1, Caspr2 and voltage-gated potassium channel antibodies in encephalitis. Nature Reviews Neurology, 2017, 13(5): 290-301.
9. Gadoth A, Pittock SJ, D Dubey, et al. Expanded phenotypes and outcomes among 256 LGI1/CASPR2-IgG positive patients. Annals of Neurology, 2017, 82(1): 79-92.
10. Patterson KR, Dalmau J, Lancaster E. Mechanisms of Caspr2 antibodies in autoimmune encephalitis and neuromyotonia. Annals of Neurology, 2018, 83(1): 40-51.
11. Bien CG, Mirzadjanova Z, Baumgartner C, et al. Anti‐contactin‐associated protein‐2 encephalitis: relevance of antibody titres, presentation and outcome. European Journal of Neurology, 2016, 24(1): 175-186.
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